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A fin-loop-like structure in GPX4 underlies neuroprotection from ferroptosis: Cell

Furthermore, oxidative stress compromises the BBB, increasing its permeability to harmful substances and promoting neuroinflammation, which amplifies oxidative damage through inflammatory cytokine release and glial activation [54,84,85,86]

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Although the brain GSH system may be activated as an adaptive response to oxidative stress, with disease duration and continued ROS overproduction, antioxidant reserves may be depleted (↓GPx, ↓GR), and neuronal metabolism may be impaired [45–47]