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If one considers that the Nrf2 transcription factor is transactivated following oxidative stress so as to stimulate autophagic flux (106,107), it is possible that taurine can attenuate the expression levels of Nrf2 transcription factor in the cerebrum of As2O3-exposed mice (103)
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however, as is discussed, the majority of these chelating agents have disadvantages such as causing cellular distribution limitations, resulting in essential metal loss and the presence of adverse side effects
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